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The crystal construction on the ligelizumab svFv:IgE-Fc3-4 complex gives specific insights into its capacity to inhibit Fc?RI and CD23 binding (Fig. 2g–k). Views on the IgE:ligelizumab and IgE:Fc?RI? complexes alongside the IgE dimer axis present that the two ligelizumab scFvs mimic the preparations of Fc?RI and IgE C?2 domains on possibly aspec